Health Blog | I Give You Health

Article by Alicia Stock

Endometrial cancer is one of the almost popular cancers in American women. In endometrial cancer, cancer cells produce in the lining of the womb. Why these cancer cells produce is not completely known. However, scientists think that estrogen levels beat a character in the growth of endometrial cancer. Endometrial cancer may develop in breast cancer patients who have been treated with tamoxifen. Women taking estrogen alone have an increased risk of developing endometrial cancer. Taking estrogen in combination with progesterone does not increase a woman’s risk of this cancer. Endometrial cancer is sometimes called uterine cancer, but there are other cells in the uterus that can become cancerous – such as muscle or myometrial cells. These form much less common cancers called sarcomas and account for less than 5 percent of uterine cancers.

Endometrial cancer is frequently detected at an earlier phase because it often produces vaginal hemorrhage between menstrual periods or after menopause. Some of the same danger factors for bosom cancer and ovarian cancer too increase the danger of endometrial cancer. Endometrial cancer usually begins in the endometrium, the lining of the uterus – a hollow, pear-shaped pelvic organ where fetal development occurs. If discovered early, this slow-growing cancer is likely to be confined to the uterus. White women are more likely to develop endometrial cancer, but black women are much more likely to die of the disease. Diabetes is a risk factor for endometrial cancer mainly because obesity and type 2 diabetes often go hand in hand. Estrogen stimulates growth of the endometrium. Replacing estrogen alone after menopause may increase the risk of endometrial cancer.

Endometrial cancer frequently develops over a period of years. Most cases of endometrial cancer produce in postmenopausal women, whose periods have stopped. Rarely does endometrial cancer hit a sophisticated phase before any signs and symptoms are existing. Conversely, some women who develop endometrial cancer appear to have no risk factors for the disease. Because endometrial cancer is usually diagnosed in the early stages, there is a better probable outcome associated with it than with other types of gynecological cancers such as cervical or ovarian cancer. Use of oral contraceptives can reduce endometrial cancer risk even as long as 10 years after you stop taking them. The risk is lowest in women who take oral contraceptives for many years. A woman with advanced endometrial cancer may have other symptoms, such as losing weight without trying.

Surgery is the almost popular main handling for endometrial cancer. Surgical handling should comprise of, at least, cytologic sample of the peritoneal fluid, abdominal exploration, palpation and biopsy of suspicious lymph nodes, abdominal hysterectomy, and removal of both ovaries. If the cancer has spread to other parts of your body, synthetic progestin, a form of the hormone progesterone, may stop it from growing. Chemotherapy is the use of drugs to kill cancer cells. In some cases, your doctor may recommend chemotherapy for endometrial cancer. These drugs enter your bloodstream and then travel through your body, killing cancer cells outside the uterus. Abdominal hysterectomy is recommended over vaginal hysterectomy because it affords the opportunity to examine and obtain washings of the abdominal cavity to detect any further evidence of cancer.

About the Author

Alicia Stock writes articles for women problems. She also writes articles for long hairstyles and japanese hairstyles.

Mike Adams from NaturalNews.com, reporting for Infowars.com visits the Burzynski research institute and speaks with Dr. Stanislaw Burzynski, founder of the research institute and clinic. Dr. Burzynski talks about how his invention of gene-targeted cancer medications called Antineoplastons has shown much greater success rates for ridding the body of cancer than any conventional method. Dr. Burzynski also explains that while his invention should be applauded by the medical industry as a whole, it actually made him a target of scrutiny and many indictments.

The term diabetes comes from the Greek language, and was coined by Aretaeus of Cappadocia. It meant “a compass, siphon.” The sense “siphon” gave rise to the use of diabetes as the name for a disease involving the emancipation of excessive amounts of urine. Diabetes is first recorded in English as diabete. It was in 1675 that Thomas Willis added the word mellitus, from Latin, meaning “honey”, to this name, as a reference to the sweet taste of the urine. This sweet taste had been noticed in urine by the ancient Greeks, Egyptians, Chinese, and Indians. It was confirmed that it was due to an excessive amount of a kind of sugar in the blood and urine of people suffering from diabetes. The ancient Indians tested for diabetes by monitoring if ants were attracted to a person’s urine, and called the ailment “sweet urine disease”.

Diabetes mellitus, quite often simply called diabetes, is a condition typified by abnormally high blood sugar and disordered metabolism. It results from low levels of insulin, a hormone present in the pancreas, with or without abnormal resistance to insulin’s effects. The disorder is characterized by disproportionate urine production, excessive thirst, and abnormal increase in appetite and fluid intake, paired with blurred vision, unexplained weight loss and indolence. These symptoms may not exist if the blood sugar is only mildly elevated. They typically worsen over days to weeks. The World Health Organization acknowledges three main forms of diabetes mellitus: type 1, type 2, and gestational diabetes, which occurs during pregnancy. Each of these types has different causes and mass allocations. Eventually however, all forms are caused due to the pancreatic beta cells being unable to produce sufficient insulin. Type 1 diabetes is usually due to reflexive annihilation of these pancreatic beta cells. Type 2 diabetes is set apart by insulin resistance in a particular target tissue that causes a demand for abnormally high amounts of insulin, which the beta cells cannot meet. Gestational diabetes is quite akin to type 2 diabetes in a way, for it involves insulin resistance; the hormones of pregnancy can cause insulin resistance in women who happen to be hereditarily predisposed to developing this condition.

Gestational diabetes typically resolves with delivery of the child, but types 1 and 2 diabetes are chronic ailments. All types have been curable since insulin became medically available in 1921. Type 1 diabetes, in which insulin is not secreted by the pancreas, is directly treatable only by injecting insulin, although dietary and other lifestyle adjustments are required. Type 2 may be managed with a permutation of dietary therapy, tablets, injections and insulin supplementation

Both type 1 and type 2 diabetes are at least partly inherited. Type 1 diabetes, although inherited, appears to be triggered by some infections, or in a less common group, by stress or environmental exposure to chemicals and drugs. There is a stronger inheritance archetype for type 2 diabetes.

Diabetes can cause many severe complications, which may occur if the disease is not properly controlled. Serious long-term complications include chronic renal failure, cardiovascular disease, nerve damage, retinal damage and micro vascular damage, which may cause impotence and poor healing of wounds – particularly of the feet, which can lead to gangrene, and which may require amputation. Adequate treatment of diabetes, as well as increased emphasis on blood pressure control and lifestyle factors, may improve the risk profile of most aforementioned complications. In the urbanized world, diabetes is probably the most important cause of adult blindness in relatively young people and non-traumatic amputation in adults. Despite the availability of treatment, diabetes has remained a major cause of death.

www.therenegadehealthshow.com – I get a lot of questions about raw food… I feel that I’m extremely qualified to answer them to a certain degree, since we practice a high raw diet. But, I also know my limits… I can’t talk about how to succeed on a 100% raw food diet for an extended period of time because I’m not in that particular category. This show will speak specifically on the raw food diet, where to start, when I pass the torch and give some information about erroneous citations of studies (that is rampant in the community… to be fair, ANY community!) to back up the 100% raw food diet. You’ll see what I’m talking about when you watch the show… go ahead and do that now…